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“PANDEMIC” is the word for 2020 according to the Merriam–Webster dictionary. No wonder, as the global COVID-19 pandemic has transformed our entire society and exposed some of the darker aspects of human nature. There is now well-established research showing that the pandemic has exaggerated and amplified pre-existing structural biases and systemic inequities in health systems. Many of the structural drivers of health-related behaviors and outcomes such as racism have been broadly characterized, including the way the COVID-19 pandemic exacerbates these drivers. But far less attention has been paid to the role of organizational and systemic factors related to goodness. The COVID-19 pandemic is revealing the unsettling truth about the issue of goodness in health. Health care workers experienced significantly more COVID-19-related bullying than people who did not work in health care settings after controlling for confounding effects, according to a study from 173 countries. Another stud

BETWEEN COVID-19, influenza, and respiratory viruses, doctors must now keep tabs on their patients at risk as the number of syphilis continues to rise in vulnerable communities, leading to calls for increased screening in those groups. According to National Infectious Disease Surveillance Report From 30 May to 12 June 2022, there was a “continuous epidemic” occurring in men who have sex with men (MSM), mainly aged 20–39 years, in urban areas, in women aged 20–39 years (both Aboriginal and Population). Torres Strait Islander and non-Indigenous) in urban areas, and Aboriginal and Torres Strait Islander peoples in northern and central Australia. “This is a very significant increase. Syphilis is a serious infection and we need to take it very seriously,” said Professor Christopher Fairley, Director of the Melbourne Center for Sexual Health and Professor of Public Health at Monash University. Syphilis is a bacterial sexually transmitted infection caused by Treponema pallidum . There are f

A study of the herpes virus infecting chickens offers new insight into the potentially problematic interactions between vaccines made from live viruses and viruses that are meant to be thwarted. Reported in the journal Virulence, the study offers direct evidence that vaccines and viruses can infect the same cells in live animals and share the molecular tools that allow the virus to infect other animals – in this case, chickens. The study focused on Marek’s disease, a viral infection that is spread when a chicken inhales flakes of dead skin or feather tissue from an infected chicken. “We’ve been trying to understand how the virus spreads from one host to another,” said University of Illinois Urbana-Champaign pathobiology professor Keith Jarosinski, who led the study. “Not only did we do it for the benefit of chickens in the poultry industry, but also because of a very similar mechanism used by the virus that causes chickenpox, where it enters through the respiratory tract and infects l